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DiGeorge syndrome, also known as 22q11.2 deletion syndrome, is a multisystem developmental disorder caused by microdeletion of chromosome 22q11.2. It primarily affects structures derived from the 3rd and 4th pharyngeal pouches, resulting in cardiac, craniofacial, immunological, endocrine, and neurodevelopmental abnormalities.

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https://www.youtube.com/watch?v=7DhBLgh0kds

CATCH-22: Mnemonic for features

Letter Feature
C Cardiac defects (especially conotruncal)
A Abnormal facies
T Thymic hypoplasia/aplasia → immune deficiency
C Cleft palate or velopharyngeal insufficiency
H Hypocalcemia (due to parathyroid hypoplasia)
22 Chromosome 22q11.2 microdeletion

Genetics


Clinical Features


Domain Feature
C: Cardiac Primarily conotruncal defects:
• Interrupted aortic arch (Type B)
• Truncus arteriosus
• Tetralogy of Fallot (ToF)
• Ventricular septal defect
• Aortic arch anomalies
A: Craniofacial (abnormal facies) **• Hypertelorism
• Short palpebral fissures
• Small chin (micrognathia)
• Low-set, posteriorly rotated ears
• Nasal bridge flattening**
T: Thymic hypoplasia/aplasia • Reduced or absent thymic shadow on CXR
• T-cell immunodeficiency → recurrent infections (especially viral/fungal)
**C: Palatal anomalies
(cleft palate)** • Cleft palate
• Submucosal cleft
• Velopharyngeal insufficiency → nasal speech, feeding difficulty
H: ‣ • Due to parathyroid hypoplasia
• Presents with seizures, tetany in neonates
Neurological/Developmental • Intellectual disability (mild to moderate)
• ADHD, autism spectrum disorders
• Psychiatric illness (e.g., schizophrenia in adolescence)

Diagnosis


Radiology


Modality Imaging features
CXR Absent thymic shadow in neonates
• Cardiomegaly if associated CHD
Echo First-line for evaluating conotruncal cardiac defects
• Truncus arteriosus, IAA (especially Type B), ToF, VSD, right arch
CTA/MRA Precise aortic arch anatomy (e.g., IAA Type B, aberrant subclavian)
• Aortic arch interruption, aberrant subclavian arteries, cervical aortic arch
Brain MR For developmental delay, seizures
• Mild cortical atrophy, corpus callosal thinning (non-specific)

Management